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NTSR2 agonists open up new avenues for the treatment of heart failure
 

NTSR2 agonists open up new avenues for the treatment of heart failure
 

Highlights will included the presentation of the Max Planck Startup Award of the Stifterverband and the plenary assembly featuring an expert dialogue on the future of democracy
 

Declining NAD+ levels in the aging heart weaken circadian rhythm and cardiac health

Renowned scientists, including Didier Stainier from the Max Planck Institute for Heart and Lung Research in Bad Nauheim, argue in the journal Genetics for a paradigm shift in the field of genetics. In their view, classical Mendelian genetics is not sufficient to explain complex gene effects in interaction with the environment and genetic background.

Membrane protein PEAR1 prevents the development of metastases
 

Transcription factor TAF4A preserves nuclei in muscle stem cells and initiates muscle regeneration after injury
 

GPR153 promotes inflammatory reactions in the vessel wall in response to acute damage

RBPMS and RBPMS2 regulate cell division and function during heart development

A team of researchers at the Max Planck Institute for Heart and Lung Research, led by Prof. Dr. Didier Y.R. Stainier, has been awarded a prestigious ERC Proof of Concept (PoC) grant to advance a groundbreaking RNA-based therapeutic strategy for Duchenne Muscular Dystrophy (DMD).

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