Suchergebnisse

Marass, M.; Beisaw, A.; Gerri, C.; Luzzani, F.; Fukuda, N.; Gunther, S.; Kuenne, C.; Reischauer, S.; Stainier, D. Y. R.: Genome-wide strategies reveal target genes of Npas4l associated with vascular development in zebrafish. DEVELOPMENT 146 (11), UNSP dev173427 (2019)

Boskovic, S.; Marin-Juez, R.; Jasnic, J.; Reischauer, S.; El Sammak, H.; Kojic, A.; Faulkner, G.; Radojkovic, D.; Stainier, D. Y. R.; Kojic, S.: Characterization of zebrafish (Danio rerio) muscle ankyrin repeat proteins reveals their conserved response to endurance exercise. PLOS ONE 13 (9), e0204312 (2018)

Guerra, A.; Germano, R. F. V.; Stone, O.; Arnaout, R.; Guenther, S.; Ahuja, S.; Uribe, V.; Vanhollebeke, B.; Stainier, D. Y. R.; Reischauer, S.: Distinct myocardial lineages break atrial symmetry during cardiogenesis in zebrafish. ELIFE 7, e32833 (2018)

Uribe, V.; Ramadass, R.; Dogra, D.; Rasouli, S. J.; Gunawan, F.; Nakajima, H.; Chiba, A.; Reischauer, S.; Mochizuki, N.; Stainier, D. Y. R.: In vivo analysis of cardiomyocyte proliferation during trabeculation. DEVELOPMENT 145 (14), UNSP dev164194 (2018)

Dogra, D.; Ahuja, S.; Kim, H.-T.; Rasouli, S. J.; Stainier, D. Y. R.; Reischauer, S.: Opposite effects of Activin type 2 receptor ligands on cardiomyocyte proliferation during development and repair. NATURE COMMUNICATIONS 8, 1902 (2017)

Gut, P.; Reischauer, S.; Stainier, D. Y. R.; Arnaout, R.: LITTLE FISH, BIG DATA: ZEBRAFISH AS A MODEL FOR CARDIOVASCULAR AND METABOLIC DISEASE. PHYSIOLOGICAL REVIEWS 97 (3), S. 889 - 938 (2017)

Reischauer, S.; Stone, O. A.; Villasenor, A.; Stainier, D. Y. R.: The specification of the hemato-vascular lineage: Lessons from a small fish. ACTA PHYSIOLOGICA 219, S. 17 - 17 (2017)

Sawamiphak, S.; Kontarakis, Z.; Filosa, A.; Reischauer, S.; Stainier, D. Y. R.: Transient cardiomyocyte fusion regulates cardiac development in zebrafish. NATURE COMMUNICATIONS 8, 1525 (2017)

Ahuja, S.; Dogra, D.; Stainier, D. Y.R.; Reischauer, S.: Id4 functions downstream of Bmp signaling to restrict TCF function in endocardial cells during atrioventricular valve development. DEVELOPMENTAL BIOLOGY 412 (1), S. 71 - 82 (2016)

Orr, N.; Arnaout, R.; Gula, L. J.; Spears, D. A.; Leong-Sit, P.; Li, Q.; Tarhuni, W.; Reischauer, S.; Chauhan, V. S.; Borkovich, M. et al.; Uppal, S.; Adler, A.; Coughlin, S. R.; Stainier, D. Y.R.; Gollob, M. H.: A mutation in the atrial-specific myosin light chain gene (MYL4) causes familial atrial fibrillation. NATURE COMMUNICATIONS 7, 11303 (2016)

Reischauer, S.; Stone, O. A.; Villasenor, A.; Chi, N.; Jin, S.-W.; Martin, M.; Lee, M. T.; Fukuda, N.; Marass, M.; Witty, A. et al.; Fiddes, I.; Kuo, T.; Chung, W.-S.; Salek, S.; Lerrigo, R.; Alsio, J.; Luo, S.; Tworus, D.; Augustine, S. M.; Mucenieks, S.; Nystedt, B.; Giraldez, A. J.; Schroth, G. P.; Andersson, O.; Stainier, D. Y.R.: Cloche is a bHLH-PAS transcription factor that drives haemato-vascular specification. NATURE 535 (7611), S. 294 - + (2016)